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JYMS : Journal of Yeungnam Medical Science

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Yee Tae Park 3 Articles
A Case of Dissection in Marfan Syndrome with Ascending Aortic Aneurysm.
Bong Young Yoon, Chang Heon Yang, Young Jo Kim, Bong Sup Shim, Hyun Woo Lee, Han Young Ryu, Tae Eun Jung, Yee Tae Park, Sung Sae Han
Yeungnam Univ J Med. 1989;6(1):179-184.   Published online June 30, 1989
DOI: https://doi.org/10.12701/yujm.1989.6.1.179
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AbstractAbstract PDF
The Marfan syndrome is a generalized connective tissue disease involving eye, musculoskeletal system, cardiovascular system, and inherited autosomal dominant with various expression type. The cardiovascular complications such as aortic aneurysm, aortic dissection, aortic regurgitation, mitral regurgitation and aortic dissection which usually occurs in previously normal sized aorta are poor prognostic factors. However, the aortic dissection which developed in patient with Marfan syndrome and aortic aneurysm was rare. We experienced one case of dissecting aneurysm in patient diagnosed as previous aortic aneurysm, aortic regurgitation, and Marfan syndrome, receiving successful operation.
Effects of Dimethyl Sulfoxide on the Differentiation of Myocardial and Endothelial Cells.
Dong Hyup Lee, Yee Tae Park, Sung Sae Han, Yung Chang Lee
Yeungnam Univ J Med. 1988;5(2):111-119.   Published online December 31, 1988
DOI: https://doi.org/10.12701/yujm.1988.5.2.111
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AbstractAbstract PDF
To elucidate the effects of dimethyl sulfoxide on of myocardial and endothelial cells in culture, the cells were exposed to 10% dimethyl sulfoxide in culture medium for 1 hour at 48 hours after cell isolation. The general morphology and the cytochemical reaction of marker enzymes for mitochondria and Golgi complexes were investigated. The results were summarized as follows 1. DMSO induced elongation and narrowing of the cells and increase of mitochondrial reaction in myocardial cells. 2. DMSO induced destruction and disruption of myofibrils in myocardial cells resulting in increase of contractile activities. 3. In the endothelial cells, DMSO suppressed proliferative activities but thiamine pyrophosphatase reactions were enhanced indicating increase of Golgi complex activity. 4. DMSO seemed to hamper with the adhesiveness and motility of the endothelial cells causing the decrease of the number of cells in vitro.
Clinical Study of 459 Cases Cardiovascular Surgery.
Han Young Ryu, Tae Eun Jung, Yee Tae Park, Sung Sae Han
Yeungnam Univ J Med. 1988;5(2):101-110.   Published online December 31, 1988
DOI: https://doi.org/10.12701/yujm.1988.5.2.101
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AbstractAbstract PDF
Between April, 1984 and September 1988, 459 patients underwent cardiovascular surgery at the Yeungnam University Hospital. Of these, 355 cases were open heart surgeries and 104 cases were non-open heart surgeries. There were 237 patients of acyanotic congenital cardiac anomalies, 40 patients of cyanotic congenital cardiac anomalies, and 85 patients of acquired heart diseases. The sex ratio of cardiovascular diseases was represented as 1:1.3 in male and female. The age distribution was ranged from 1 day to 65 years old. The common congenital cardiovascular anomalies were ventricular septal defect (38.7%), patent ductus arteriosus (25.5%), atrial septal defect (20.7%), Tetralogy of Fallot (8.3%), and pulmonary stenosis (2.4%) in order of frequency. Among 87 acquired cardiovascular diseases, 81 patients underwent operation for cardiac valvular lesions, 51 patients had mitral valve replacement and 13 patients had aortic valve replacement and 17 patients had double valve replacement. The overall mortality of cardiovascular surgery was 3.3% and mortality of open heart surgery was 3.9%.

JYMS : Journal of Yeungnam Medical Science